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        "resumen" => "<span class="elsevierStyleSectionTitle">Introducci&#243;n</span><p class="elsevierStyleSimplePara elsevierViewall">El s&#237;ndrome de transfusi&#243;n fetofetal presenta una importante morbimortalidad&#44; en parte debida a las alteraciones cardiovasculares que genera&#46;</p> <span class="elsevierStyleSectionTitle">Casos cl&#237;nicos</span><p class="elsevierStyleSimplePara elsevierViewall"><span class="elsevierStyleItalic">Paciente 1</span>&#46; Se realiza ces&#225;rea en la semana 28 por disfunci&#243;n mioc&#225;rdica e insuficiencia tricusp&#237;dea grave en el feto receptor&#46; A las 12 h de vida&#44; el feto receptor desarrolla un shock refractario&#46; Se objetiva hipertrofia biventricular grave&#44; que acaba en fallecimiento a las 60 h de vida en fallo multiorg&#225;nico&#46;</p><p class="elsevierStyleSimplePara elsevierViewall"><span class="elsevierStyleItalic">Paciente 2</span>&#46; Se practica ces&#225;rea en la semana 29 por cardiomegalia e insuficiencia card&#237;aca congestiva&#46; Al nacer presenta mala perfusi&#243;n perif&#233;rica e hipertensi&#243;n&#46; Se objetiva hipertrofia biventricular que progresa hasta generar una estenosis medioventricular derecha ligera a las 72 h&#46; Se inicia regresi&#243;n al octavo d&#237;a y persiste ligera hipertrofia a los 6 meses&#46;</p> <span class="elsevierStyleSectionTitle">Discusi&#243;n</span><p class="elsevierStyleSimplePara elsevierViewall">Las alteraciones card&#237;acas en el s&#237;ndrome de transfusion fetofetal aparecen s&#243;lo en los fetos receptores&#46; Los hallazgos m&#225;s frecuentes son la existencia de dilataci&#243;n&#44; hipertrofia&#44; disfunci&#243;n mioc&#225;rdica&#44; insuficiencia tricusp&#237;dea y estenosis pulmonar&#46; Estas alteraciones suelen regresar con los meses&#44; pudiendo tambi&#233;n empeorar&#46;</p>"
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        "resumen" => "<span class="elsevierStyleSectionTitle">Introduction</span><p class="elsevierStyleSimplePara elsevierViewall">Twin-twin transfusion syndrome carries high morbidity and mortality&#44; related in part to the cardiovascular alterations that it produces&#46;</p> <span class="elsevierStyleSectionTitle">Case report</span><p class="elsevierStyleSimplePara elsevierViewall"><span class="elsevierStyleItalic">Patient 1</span> underwent a cesarian section at week 28 due to myocardial dysfunction and severe tricuspid regurgitation in the recipient twin&#46; Twelve hours after birth the recipient twin developed refractory shock&#46; Severe biventricular hypertrophy was observed and the twin died 60 hours after birth from multisystemic failure&#46;</p><p class="elsevierStyleSimplePara elsevierViewall"><span class="elsevierStyleItalic">Patient 2</span> underwent a cesarian section at week 29 due to cardiomegaly and cardiac insufficiency in the recipient twin&#46; At birth&#44; he showed hypertension and poor peripheral perfusion&#46; Biventricular hypertrophy was observed&#44; which increased until it produced a mild right midventricular stenosis at 72 hours&#44; decreasing after the 8th day&#46; Mild hypertrophy persisted at 6 months of life&#46;</p> <span class="elsevierStyleSectionTitle">Discussion</span><p class="elsevierStyleSimplePara elsevierViewall">Cardiovascular alterations appear only in recipient fetuses affected by twin-twin transfusion syndrome&#46; The most frequent findings are cardiac dilation&#44; hypertrophy&#44; myocardial dysfunction&#44; tricuspid regurgitation and pulmonary stenosis&#46; These alterations usually disappear during the first few months of life&#44; but may also worsen&#46;</p>"
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Notas clínicas
Transfusión fetofetal y complicaciones cardiovasculares
Twin-twin transfusion syndrome and cardiovascular complications
P. Betrián Blasco
Autor para correspondencia
pedrobetrian@yahoo.es

Correspondencia: Dr. P. Betrián Blasco. Fraga, 18, 3.° C. 50003 Zaragoza. España.
, J. Girona Comas, D.C. Albert Brotons
Servicio de Cardiología Pediátrica. Hospital Maternoinfantil Vall d’Hebron. Barcelona. España
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            1 => "Hipertrofia"
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            3 => "Embarazo gemelar"
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        "resumen" => "<span class="elsevierStyleSectionTitle">Introducci&#243;n</span><p class="elsevierStyleSimplePara elsevierViewall">El s&#237;ndrome de transfusi&#243;n fetofetal presenta una importante morbimortalidad&#44; en parte debida a las alteraciones cardiovasculares que genera&#46;</p> <span class="elsevierStyleSectionTitle">Casos cl&#237;nicos</span><p class="elsevierStyleSimplePara elsevierViewall"><span class="elsevierStyleItalic">Paciente 1</span>&#46; Se realiza ces&#225;rea en la semana 28 por disfunci&#243;n mioc&#225;rdica e insuficiencia tricusp&#237;dea grave en el feto receptor&#46; A las 12 h de vida&#44; el feto receptor desarrolla un shock refractario&#46; Se objetiva hipertrofia biventricular grave&#44; que acaba en fallecimiento a las 60 h de vida en fallo multiorg&#225;nico&#46;</p><p class="elsevierStyleSimplePara elsevierViewall"><span class="elsevierStyleItalic">Paciente 2</span>&#46; Se practica ces&#225;rea en la semana 29 por cardiomegalia e insuficiencia card&#237;aca congestiva&#46; Al nacer presenta mala perfusi&#243;n perif&#233;rica e hipertensi&#243;n&#46; Se objetiva hipertrofia biventricular que progresa hasta generar una estenosis medioventricular derecha ligera a las 72 h&#46; Se inicia regresi&#243;n al octavo d&#237;a y persiste ligera hipertrofia a los 6 meses&#46;</p> <span class="elsevierStyleSectionTitle">Discusi&#243;n</span><p class="elsevierStyleSimplePara elsevierViewall">Las alteraciones card&#237;acas en el s&#237;ndrome de transfusion fetofetal aparecen s&#243;lo en los fetos receptores&#46; Los hallazgos m&#225;s frecuentes son la existencia de dilataci&#243;n&#44; hipertrofia&#44; disfunci&#243;n mioc&#225;rdica&#44; insuficiencia tricusp&#237;dea y estenosis pulmonar&#46; Estas alteraciones suelen regresar con los meses&#44; pudiendo tambi&#233;n empeorar&#46;</p>"
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        "resumen" => "<span class="elsevierStyleSectionTitle">Introduction</span><p class="elsevierStyleSimplePara elsevierViewall">Twin-twin transfusion syndrome carries high morbidity and mortality&#44; related in part to the cardiovascular alterations that it produces&#46;</p> <span class="elsevierStyleSectionTitle">Case report</span><p class="elsevierStyleSimplePara elsevierViewall"><span class="elsevierStyleItalic">Patient 1</span> underwent a cesarian section at week 28 due to myocardial dysfunction and severe tricuspid regurgitation in the recipient twin&#46; Twelve hours after birth the recipient twin developed refractory shock&#46; Severe biventricular hypertrophy was observed and the twin died 60 hours after birth from multisystemic failure&#46;</p><p class="elsevierStyleSimplePara elsevierViewall"><span class="elsevierStyleItalic">Patient 2</span> underwent a cesarian section at week 29 due to cardiomegaly and cardiac insufficiency in the recipient twin&#46; At birth&#44; he showed hypertension and poor peripheral perfusion&#46; Biventricular hypertrophy was observed&#44; which increased until it produced a mild right midventricular stenosis at 72 hours&#44; decreasing after the 8th day&#46; Mild hypertrophy persisted at 6 months of life&#46;</p> <span class="elsevierStyleSectionTitle">Discussion</span><p class="elsevierStyleSimplePara elsevierViewall">Cardiovascular alterations appear only in recipient fetuses affected by twin-twin transfusion syndrome&#46; The most frequent findings are cardiac dilation&#44; hypertrophy&#44; myocardial dysfunction&#44; tricuspid regurgitation and pulmonary stenosis&#46; These alterations usually disappear during the first few months of life&#44; but may also worsen&#46;</p>"
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Información del artículo
ISSN: 16954033
Idioma original: Español
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