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Vol. 58. Issue 3.
Pages 232-235 (1 March 2003)
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Vol. 58. Issue 3.
Pages 232-235 (1 March 2003)
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Presencia de genu valgum en obesos: causa o efecto
Presence of genu valgum in obesechildren: cause or effect?
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B. Bonet Serra
Corresponding author
bbjbonet@fhalcorcon.es

Correspondencia: Servicio de Pediatría. Fundación Hospital Alcorcón.Budapest, 1. 28922 Alcorcón. Madrid. España.
, A. Quintanar Rioja, M.a Alavés Buforn, J. Martínez Orgado, M. Espino Hernández, F.J. Pérez-Lescure Picarzo
Servicio de Pediatría. Fundación Hospital Alcorcón. Madrid. España
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Antecedentes

Las alteraciones corporales que puedan afectar la actividad física podrían desempeñar un papel relevante en el consumo de calorías y, por tanto, en el desarrollo de obesidad.

Objetivos

Determinar, en niños con sobrepeso, la presencia de genu valgum, alteración que puede afectar la realizaciónde ejercicio físico y disminuir el gasto calórico.

Métodos

Se estudiaron 35 niños con sobrepeso, en los que se descartaron enfermedades endocrinológicas como causa de la obesidad. Como controles, se estudiaron 29 niños sin sobrepeso de similar edad. En todos los niños se determinó la talla, el peso, el índice de masa corporal (IMC) y la distancia intermaleolar (DIM) como índice del grado de genu valgum. Las diferencias entre grupos se realizaron utilizando el test de análisis de la varianza (ANOVA) y la asociación entre diferentes variables se analizó mediante la correlación lineal de Pearson.

Resultados

Los niños con sobrepeso mostraron un IMC superior al grupo control. Este grupo también presento una DIM superior a la observada en el grupo de niños sin sobrepeso (11,0 6 0,6 frente a 2,90 6 0,43; p < 0,001). En los niñoscon sobrepeso se observó una correlación positiva entre el IMC y la DIM (p < 0,009). El 50% de los niños con sobrepeso presenta una DIM superior a los 10 cm, valor considerado patológico.

Conclusiones

En los niños obesos, la incidencia de genu valgum esmuy superior a la observada en la población sin sobrepeso de la misma edad. Dicha alteración podría limitar la actividad física y originar el desarrollo de obesidad.

Palabras clave:
Obesidad
Genu valgum
Niños
Distancia intermaleolar
Background

Changes in body configuration that may affect the physical activity may play a role in the caloric consumption and led to the development of obesity.

Objectives

To determine the presence of genu valgum, an alteration that may decrease physical activity and caloric expenditure, in overweight children.

Methods

Thirty-five overweight children without any endocrinological alterations that could lead to obesity were studied. Twenty-nine non-overweight children of a similar age were studied as a control group. In all children weight, height, and body mass index (BMI) were studied, and intermalleolar distance was used to measure the degree of genu valgum. The differences between groups were stud-ied using ANOVA and the correlation between variables was determined using Pearson's correlation.

Results

BMI was higher in overweight children than in the control group. Intermalleolar distance was greater in overweight children than in the non-overweight group (11.0 6 0.6 vs 2.90 6 0.43; p <0.001). A positive correlation between the intermalleolar distance and the BMI was ob-served in the overweight group (p < 0.009). Fifty percent of the overweight children showed an intermalleolar distance of more than 10 cm, a value considered abnormal.

Conclusions

The incidence of genu valgum is much higher in overweight children than in non-overweight children of the same age. This alteration may lead to decreased physicalactivity and lead to obesity.

Key words:
Genu valgum
Obesity
Children
Medial malleoli
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Bibliografía
[1.]
P. Bjorntorp.
Obesity.
[2.]
M. Rosembaum, R.L. Lebeil, J. Hirsch.
Obesity.
N Engl J Med, 337 (1997), pp. 396-407
[3.]
J. Hirsch, L.B. Salans, L.J. Aronne.
Obesity.
Principles and practice of endocrinology and metabolism, pp. 1239-1246
[4.]
E. Ravussin, B.A. Swinburn.
Pathophysiology of obesity.
Lancet, 340 (1992), pp. 404-413
[5.]
M. Shah, R.W. Jeffery.
Is obesity due to overeating and inactivity, or to a defective metabolic rateα A review.
Ann Beh Med, 13 (1991), pp. 73-81
[6.]
A.C. King, D.L. Tribble.
The role of exercise in weight regulation in nonathletes.
Sports Med, 11 (1991), pp. 331-349
[7.]
D.H. Ledbetter, A. Ballabio.
Molecular cytogenetics of contiguous gene syndromes: Mechanism and consequences of gene dosage imbalance.
The metabolic and molecular bases of inherited disease, pp. 811-896
[8.]
I.S. Farooqi, S.A. Jebb, G. Langmack, E. Lawrence, C.H. Cheetham, A.M. Prentice.
Effects of recominant leptin therapy in a child with congenital leptin deficiency.
N Engl J Med, 341 (1999), pp. 879-884
[9.]
H. Rau, B.J. Reaves, S. O'Rahilly, J.P. Whitehead.
Truncated human leptin (delta 133) associated with extreme obesity undergoes proteasomal degradation after defective intracellular transport.
Endocrinology, 140 (1999), pp. 1718-1723
[10.]
G.R. White, G.A. Mencio.
Genu valgum in children: Diagnostic and therapeutic alternatives.
J Am Acad Orthop Surg, 3 (1995), pp. 275-283
[11.]
R.D. Neddlman.
Growth and development.
Nelson's Textbook of Pediatrics, 16th, pp. 51-52
[12.]
M. Garaulet, A. Martinez, F. Victoria, F. Perez-Llamas, R.M. Ortega, S. Zamora.
Difference in dietary intake and activity level between normal-weight and overweight or obese adolescents.
J Pediatr Gastroenterol Nutr, 30 (2000), pp. 253-258
[13.]
C.S. Berkey, H.R. Rockett, A.E. Field, M.W. Gillman, A.L. Frazier, C.A. Camargo.
Activity, dietary intake, and weight changes in a longitudinal study of preadolescent and adolescent boys and girls.
Pediatrics, 105 (2000), pp. 56
[14.]
K. Samaras, P.J. Kelly, M.N. Chiano, T.D. Spector, L.V. Campbell.
Genetic and environmental influences on total-body and central abdominal fat: The effect of physical activity in female twins.
Ann Intern Med, 130 (1999), pp. 873-882
[15.]
R.L. Weinsier.
Genes and obesity: Is there reason to change our behaviors.
Ann Intern Med, 130 (1999), pp. 938-939
[18.]
C.S. Fox, J. Esparza, M. Nicolson, P.H. Bennett, L.O. Schulz, M.E. Valencia.
Is a low leptin concentration, a low resting metabolic rate, or both the expression of the "Thrifty genotype"α Results form Mexican Pima Indians.
Am J Clin Nutr, 68 (1998), pp. 1053-1057
[17.]
J.R. Davids, M. Huskamp, A.M. Bagley.
A dynamic biomechanical analysis of the etiology of adolescent tibia vara.
J Pediatr Orthop, 16 (1996), pp. 461-468
Copyright © 2003. Asociación Española de Pediatría
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