Elsevier

The Lancet

Volume 345, Issue 8943, 21 January 1995, Pages 197-198
The Lancet

Letters to the Editor
HPV-16 transmission

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    Confirmation of the perinatal transmission in different mucosa (genital, oral) was subsequently supported in several studies, although the exact mode of transmission is not well understood. Previous small studies have reported widely varying rates of infection in newborns, with estimates ranging from 4% to 79% among day old infants born to mothers testing positive for HPV during pregnancy [12–24]. There is a need for a better understanding of the rate and determinants of perinatal transmission as well as the rate and determinants of HPV persistence in children in a prospective study with a large sample size and long term follow-up [25].

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    It is during this differentiation process (vegetative phase) that the viral genome replicates to a higher copy number and the capsid proteins are expressed and that virions are assembled and eventually shed.44 Mucosal HPVs are commonly transmitted by close contact, in particular sexual contact, although other routes of infection have been documented.47–49 Most individuals (80%–85%) acquire an HPV infection at some stage in their lives, generally in childhood for cutaneous types (hand and plantar warts) and in early adulthood for mucosal types by sexual transmission.

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